Barretts Esophagus | Pathogenesis | Gastroenterology Lectu

VIDEO DESCRIPTION:

Play, download and edit the free video Barretts Esophagus | Pathogenesis | Gastroenterology Lectures | V-Learning | sqadia.com.

This sqadia.com medical video lecture is all about Barrett’s esophagus. In this V learning lecture, pathogenesis, epidemiology, and endoscopic comparison of normal esophagus and Barrett’s esophagus is discussed in conjunction with complications, molecular biology of neoplasia and treatment.

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Lecture Duration - 00:49:03
Release Date - May 2020

Watch complete lecture on sqadia.com
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PATHOGENESIS
Several physiological abnormalities such as extreme hypotension of lower esophageal sphincter, ineffective esophageal motility, and dudenogastric reflux are conversed about initially along with their potential consequences. After that genes involved in Barrett’s metaplasia are pursued. Moreover, characteristics of Barrett’s epithelial cells are presented.

EPIDEMIOLOGY
Barrett’s esophagus is diagnosed in 55 years old people. Epidemiologically, it is prevalent between 1.6% and 6.8%, and uncommon in Asian populations. Then Dr. Hamna Iftikhar talks about the risk of cancer in patients with non-dysplastic Barrett’s esophagus. Subsequently, protective role of Helicobacter Pylori infection is discussed.

ENDOSCOPY AND BIOPSY
At first, medical practitioner delineates the endoscopic comparison of normal esophagus and Barrett’s esophagus. Following this, endoscopy with biopsy is considered. Intestinal type and Gastric cardiac type are the two histological types of epithelium discussed over here. Then classification according to segments is given which encompasses long segment, short segment, and Prague C and M criteria.

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COMPLICATIONS
At the beginning of this section, molecular biology of neoplasia is elaborated. Then light is shed on aneuploidy which is a biomarker for neoplastic progression in Barrett’s esophagus. Dr. Hamna Iftikhar also explains how aneuploidy can be detected. Subsequently, dysplasia is discussed. Lastly, incidence of esophageal cancer is reported.

TREATMENT
In the beginning, treatment of GERD is discussed. Then information about aspirin and NSAIDs is given that how these drugs are protective against esophageal adenocarcinoma. Conclusively, focus is shifted towards treatment of mucosal neoplasia which include endoscopic ablative therapy, endoscopic mucosal resection, endoscopic eradication therapy, and esophagectomy.

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